STUDY OF LIVER HISTOPATHOLOGY OF WISTAR RATS EXPOSED TO HEAVY METALS

LIVER HISTOPATHOLOGY OF WISTAR RATS EXPOSED TO HEAVY METALS

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LIVER HISTOPATHOLOGY OF WISTAR RATS EXPOSED TO HEAVY METALS

Background

Heavy metal exposure is a critical environmental and public health concern due to its hepatotoxic potential (Tchounwou et al., 2012). Metals such as cadmium (Cd), lead (Pb), mercury (Hg), and arsenic (As) can accumulate in liver tissue, causing oxidative stress, inflammation, fibrosis, and necrosis (Jaishankar et al., 2014). The liver, as the primary detoxifying organ, is particularly vulnerable to these toxicants (El-Sharaky et al., 2012).

Animal models, especially Wistar rats, are widely used to investigate the pathophysiological mechanisms of heavy metal toxicity due to their genetic uniformity and well-characterized physiology (Abdel-Wahab et al., 2016). Histopathological assessment provides crucial insights into cellular and tissue-level changes induced by metal exposure, complementing biochemical and molecular analyses.

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